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congenital hypertrophic
pyloric stenosis
(CHPS)
* Incidence:
e It is the commonest cause in surgical practice for persistent
vomiting in infancy.
e 1 in every 400 live births, more 1%. born infant , more in males
(4:1) & may be familial.
* Aetiology: Immaturity of the ganglion cells — failure or delayed
relaxation of pyloric sphincter.
* Pthology:
1. Pylorus. Hypertrophy of the circular muscle layer of the pyloric
canal which stops abruptly at the duodenum and tapers towards the
body. The pylorus projects into the duodenum forming a duodenal
fornix around it.
2. Stomach: first there is thickening & hypertrophy of gastric wall but
later on the stomach is markedly dilated.
3. Intestine is empty and collapsed.
To esophagus —
.
Kx (rnte\
Cardia \
\
a
:|
incisura anguiarig 2)
+)
To duodenum ge — Fi
Pyloric > é
—
sphincter
Pyloric antrum
Sulcus intermedius
PAEDIATRIC SURGERY
congenital hypertrophic
pyloric stenosis
(CHPS)
* Incidence:
e It is the commonest cause in surgical practice for persistent
vomiting in infancy.
e 1 in every 400 live births, more 1%. born infant , more in males
(4:1) & may be familial.
* Aetiology: Immaturity of the ganglion cells — failure or delayed
relaxation of pyloric sphincter.
* Pthology:
1. Pylorus. Hypertrophy of the circular muscle layer of the pyloric
canal which stops abruptly at the duodenum and tapers towards the
body. The pylorus projects into the duodenum forming a duodenal
fornix around it.
2. Stomach: first there is thickening & hypertrophy of gastric wall but
later on the stomach is markedly dilated.
3. Intestine is empty and collapsed.
To esophagus —
.
Kx (rnte\
Cardia \
\
a
:|
incisura anguiarig 2)
+)
To duodenum ge — Fi
Pyloric > é
—
sphincter
Pyloric antrum
Sulcus intermedius
* Complications: Repeated vomiting leads to:
1.Loss of acid + metabolic alkalosis — tetany.
2.Hyponatraemia , hypokalaemia & hypocalcaemia due to Na, K &
calcium loss respectively.
3. Dehydration.
4. Anaemia & loss of weight.
5. Respiratory obstruction & infection due to inhalation of vomitus .
* Clinical Picture:
1. The condition usually manifests itself 2-4 weeks after birth.
2. The infant is constantly crying due to constant colic and hunger.
3. Vomiting: is most important prominent feature, repeated,
projectile, never bile stained, contains food of previous meals or
days, foul odour (Fermentation).
4. Progressive constipation with small hard rabit stool and
decreasing urination.
5. Picture of complications , dehydration ,weight loss & anaemia .
6. Thick pyloric canal is felt in 90% of cases as olive, oval, firm
mass at the level of L1 vertebra just to right side of middle line is
diagnostic.
6. Epigastric fullness with visible peristaltic waves in the
epigastrium propagating from left to right.
7. In 10% of cases haematemesis may occur due to gastritis &
oesophagitis.
¢~- N.B: Any neonate presenting with repeated projectile, non
bile stained vomiting, associated hunger & firm stools
should by considered to have CHPS until proved otherwise.
Caterpillar sign
* Treatment: Only surgical.
e Rapid preoperative preparation :
> I.V fluids to correct fluid and electrolyte imbalance.
> Nasogastric tube for gastric suction and gastric wash.
> Treatment of respiratory infections.
e Exposure :
> Laparoscopic approach.
> Ramstedt’s Operation: Right upper quadrant transverse
incision.
> Method :
# Pyloromyotomy: The thick circular muscle layer in pyloric
region is incised longitudinal down to the submucosa , stopping
short of prepyloric vein > pyloric mucosa bulges.
Ramstedt’s Pyloromyotomy
te
Fia. 2: Pvlorus being spread out until mucosa bulaes